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1. Adams, W. J., and Morris, D. L. Short-course cimetidine and survival with colorectal cancer. Lancet, 344: 1768 1769, Matsumoto, S. Cimet9dine and survival with colorectal cancer. Lancet, 346: 115, 1995. Svendsen, L. B., Ross, C., Knigge, U., Frederiksen, H. J., Graversen, P., Kjaergard, J., Luke, M., Stimpel, H., and Sparso, B. H. Icmetidine as an adjuvant treatment in colorectal cancer. Dis. Colon Rectum, 38: 514 518, Hellstrand, K., Naredi, P., Lindner, P., Lundholm, K., Rudenstam, C. M., Hermodsson, S., Asztely, M., and Hafstrom, L. Histamine in immunotherapy of advanced melanoma. Cancer Immunol. Immunother., 39: 416 419.
Cimetidine dose in dogs29 Cholestyramine and colestipol These are drugs used to reduce cholesterol levels and they may bind to some herbs forming an insoluble complex thus decreasing the absorption of both substances because the size of the insoluble complex is too large to pass through the intestinal wall.xlii Antacids Antacid preparations change the pH of the stomach and may therefore interfere with the absorption of herbs. Drugs such as cimetidine Tagamet ; , ranitidine Zantac ; and omeprazole Losec ; inhibit the secretion of stomach acids and therefore herbs may not be broken down properly, leading to poor absorption in the intestines. xliii This interaction can be avoided simply by taking the herbs separately from these drugs by at least two hours. Drugs that inhibit liver metabolism Some drugs slow down or inhibit liver metabolism: examples are cimetidine Tagamet ; , erythromycin, ethanol, fluconazole Diflucan ; , itraconazole Sporanox ; and ketoconazole Nizoral ; . These drugs slow down liver metabolism and therefore herbs active ingredients will be inactivated more slowly and their overall effectiveness may be prolonged: for this reason, if the patient is taking any of the above drugs, we may need to lower the dosage of the herbs.xliv Drugs that inhibit kidney excretion Any slowing down of kidney excretion will lead to an accumulation of herbs and drugs ; in the body. Drugs that tend to damage the kidneys include methotrexate, tobramicin and gentamicin: as a safety precaution, if the patient is taking these drugs, it may be necessary to lower the dose of the herbs.xlv Diuretic drugs If the patient is taking diuretic drugs, diuretic herbs e.g. Fu Ling, Zhu Ling, Ze Xie, etc. ; should be used with caution and their dosage adjusted as their action may potentiate that of the drugs.KEY WORDS histamine; maze learning; dizocilpine; memory disorders ABSTRACT AIM: To investigate whether or not histamine is involved in spatial memory deficits induced by dizocilpine MK801 ; as evaluated by 8-arm radial maze of rats. METHODS: 8-Arm 4-arm baited ; radial maze was used to measure spatial memory in rats. RESULTS: Bilaterally intrahippocampal ih ; injection of MK-801 0.3 g site ; impaired working memory and reference memory in rats. Both histamine 50, 100 ng site, ih ; and intraperitoneal ip ; injection of histidine 100, 200 mg kg ; markedly improved the spatial memory deficits induced by MK-801. On the other hand, the ameliorating effect of histidine 100 mg kg, ip ; was completely antagonized by fluoromethylhistidine -FMH, 5 g site, ih ; , a potent and selective histidine decarboxylase HDC ; inhibitor, and H1-antagonist pyrilamine 1 g site, ih ; , but not by H2-antagonist cimetidine, even at a high dose 2.5 g site, ih ; . CONCLUSION: The hippocampal histamine plays an important role in the ameliorating effect on MK-801-induced spatial memory deficits, and its action is mediated through postsynaptic H1-receptor and differin. Pittsburgh-an offer that was too good to refuse. I was given the opportunity to direct the PICU there, an exciting and somewhat scary opportunity one year out of fellowship. You should understand that the primary reason I was made that offer was that there were barely a dozen people senior to me in the field-and they were all happy where they were. Most of them are the previous recipients of this award. Their contentedness in their respective institutions gave me the opportunity of a lifetime. In addition, I was joining what was arguably the strongest intensive care community in the world, completely adult-focused at that time to be sure, but anxious to support pediatric intensive care as well. Again, my naivete about the people around me was stupendous-I came to understand the department's role in history only later. Peter Safar, Ake Grenvik, Peter Winter, and others had long since established a world-class critical care program and welcomed me and my ideals to do the same for pediatrics-with enthusiasm and support that never waned. I have now been in Pittsburgh for 22 years, and it has been a very rich two decades. I've already mentioned the astonishing and wonderful community I entered in 1981. Nonetheless, Pittsburgh was a sleepy medical community in many ways at that time-an excellent clinical center, but not an academic powerhouse overall. I arrived a few months after Dr. Thomas Starzl started a liver transplantation program-he'd thought he'd do 10-20 liver transplants a year, to start, but in the first year of the program, he did 50 and soon we'd reached 120 a year. On the adult side they were doing 4-5 times that many. Pittsburgh became a national "phenom"- and the needs of these patients spawned growth in many other specialties and subspecialties. There was money for research in clinical and basic science. Success in one area promoted success in many others. Even after transplantation became a routine part of modern medicine, moved on to other institutions, and our numbers became much more reasonable, the institution had been transformed into a world-class academic medical center. We in Pediatric Critical Care helped make it happen and were the beneficiaries of the transformation: the unit grew from 10 to 16 now a 59 bed complex. The faculty expanded from 2 to 10; the fellows from 2 to 11. With the growth of clinical activity we could attract new faculty-with new faculty came the time, talent, and training to develop a research program. With clinical and bench research has come improved patient care. I have had the good fortune of recruiting and working with a.
| Cimetidine verruca vulgarisInflamed airways become hypersensitive to a variety of stimuli, including dust mites, animal dander, pollen, air pollution, cigarette smoke, medications, weather conditions, and exercise and eldepryl, for example, cimetidine over the counter. Dr. Billings is a certified staff anaesthetist and pain management consultant. He had pain clinics in four hospitals and teaches medical and surgical Residents. He worked as a Fellow under Dr. Gale at the end of his training as an anaesthetist at the teaching hospital. With reference to the patient 1's resuscitation, he said that it was difficult for him to gather the facts of all that occurred. In his evidence-in-chief, he stated that he would not push the team leader of a resuscitation aside because he had to respect the finding of a trained colleague. However, responding to a hypothetical situation, Dr. Billings testified that if the nurses did not feel a pulse but the team leader said that there was a pulse and he was doubtful about the presence of a pulse, he would go and feel the pulse for himself. If he did not feel a pulse and there was no blood pressure being recorded, he testified that he would definitely start CPR. On cross-examination, he agreed that the resuscitation was not done properly and did not meet the expected standard. Nurse Donna Allerton is a qualified ACLS Course Director and instructor and for 4 to 5 years taught this course at McMaster University to physicians, nurses and paramedics. She testified that if a patient is not responsive, one should call for help, check to see if the patient is breathing, then check for a pulse and if no pulse is found, one should begin CPR. She agreed that if the patient has a pulse, there should be recordable blood pressure. She added that it was Dr. B who was in charge of the patient and thus was the team leader of the code and it was also his patient. Dr. B had also most recently repeated his ACLS certification. She agreed that Dr. Gale was helping with the resuscitation in attending to airway breathing and the intubated patient and that Dr. B was attending to the circulation of the patient.Cimetidine jmol kg-' ; Fig. 3. Effects of increasing doses of cimetidine on secretory response of rabbit pancreas. The secretory rates for pancreatic juice flow and the outputs of inorganic anions and enzymes are expressed as percentage change with respect to control experiments. Each point is the mean + S.E. of mean; n 6. Horizontal bars indicate secretin-CCK saline infusion. Asterisks indicate significant difference from control values. * P 0 05; * P 0-001 and feldene. |
Agents that dissolve blood clots antacids antiinflammatory agents nsaids such as ibuprofen ; aspirin blood thinners such as warfarin cimetidine cilostazol clopidogrel cyclosporine digoxin dipyridamole fish oil omega-3 fatty acids ; supplements herbal or dietary supplements like feverfew, garlic, ginger, ginkgo biloba, and horse chestnut phenytoin prasterone, dehydroepiandrosterone, dhea supplements theophylline inform your health care professional about all other medicines you are taking, including non-prescription medicines and frusemide.
60 heightened provision of qualified or local health care workers or both has been associated with reduced crude mortality rates and under-5 mortality rates.
Acetylcholine ACh ; release from hippocampal cholinergic nerve terminals is influenced by neurotrophic factors and neuromodulators. Brain-derived neutrophic factor BDNF ; is a neurotrophin which exerts its actions through the activation of TrkB receptors. Adenosine is a neuromodulator which mainly exerts its actions through the activation of A1-inhibitory and A2A-excitatory receptors. Knipper and collaborators 1994 ; were the first to show that BDNF increases evoked [3H]ACh release from hippocampal sinaptosomes by high-K + depolarisation. On the other hand, there is evidence that A2A receptors activation is needed to observe the facilitatory actions mediated by BDNF on synaptic transmission Digenes et al. 2003 ; . We now studied the effect of BDNF on evoked [3H]ACh release from whole slices of the hippocampus by electrical stimulation and investigated if A2A receptors blockade could prevent such effect. Release of [3H]ACh from hippocampal slices pre-incubated with [3H-methyl]choline 12.3 Ci ml, 0.15 M was evoked by two sets of supramaximal rectangular pulses 2 Hz, 240 pulses each ; , S1 at minute 6 and S2 at minute 36 after starting sample collection. When testing the effect of BDNF 20 ng ml 0.8 nM ; , this neurotrophin was added 15 min before S2 and its effect was expressed as modifications of the S2 S1 ratio compared to control no drug added ; . When studying the effect of BDNF after previous blockade of A2A receptors, A2A receptor antagonist, SCH 58261 50 nM ; , was present during the whole experiment and BDNF 0.8 nM ; was added to the perfusion solution as described before. Stimulation by S1 or increased tritium release between 2 to 3.5 times over basal values. In control conditions, the average S2 S1 ratio was 0.90 0.07 n 3 ; . BDNF 0.8 nM ; significantly increased the S2 S1 ratio to 1.09 0.05 n 3 ; , which corresponds to a facilitation of [3H]ACh release of 22.3 4.0 % P 0.05 ; . This effect was significantly prevented n 2, P 0.05 ; in the presence of SCH 58261 50 nM ; % change of S2 S1 ratio caused by BDNF -5.0 7.0% ; . The results show that acute application of BDNF increases the electrically evoked [3 H]ACh release from hippocampal slices. Such effect is prevented under previous blockade of A2A adenosine receptors. A2A receptor activation seems, therefore, necessary to observe BDNF facilitation of [3H]ACh release and keflex.
Never use "U" for units. Never use chemical names. Never abbreviate drug names - MSO4 Never abbreviate once daily as OD may be read as right eye. Never abbreviate Q.D. may be read as QID Never use g, write out mcg, for instance, cimetidine suspension.
The result of the investigations outlined above was the decision to commission the "Japan Council for Quality Health Care", currently a public-service corporation, to organize the EBM databases in this country. The main reason for entrusting this task to the Japan Council for Quality Health Care was that the council is a neutral public-service corporation. However, at the same time, a number of other reasons were advanced including the fact that the "Assessments of the Quality of Health Care in Hospitals" that are currently being undertaken by the council also represents one facet of the "Medical Technology and nifedipine.
The widespread use of aciclovir may be associated with an increase in the level of resistance to aciclovir, particularly in countries where HIV prevalence is high.WHO should establish two or three sites in Africa to monitor the emergence of resistance to aciclovir, for example, cimetidine brand.
Note: This isolate was tested by the BACTEC MGIT 960, using critical lowest ; concentrations established by the United States Public Health Service and recommended by the Clinical and Laboratory Standards Institute CLSI, formerly NCCLS ; . 650 Iwilei Road, Suite 300, Honolulu, HI 96817 Phone 808 ; 589-5100 Fax 808 ; 593-8357 and reminyl.
Drug name sonata spectrazole spiriva sporanox sski starlix stelazine stimate strattera suboxone subutex sucraid sular sumycin surmontil sustiva symbyax symmetrel synalar synthroid syprine tagamet tamiflu generic name zaleplon econazole nitrate tiotropium bromide itraconazole potassium iodide saturated solution nateglinide trifluoperazine desmopressin acetate atomoxetine hcl buprenorphine hcl naloxone hcl buprenorphine hcl sacrosidase nisoldipine tetracycline hcl trimipramine maleate efavirenz olanzapine fluoxetine hcl amantadine fluocinolone levothyroxine trientine hcl cimetidine oseltamivir phosphate mc * f f notes limit of 1 day and 60 capsules year!
Ranitidine interacts differently than cietidine and only minimally 10% ; inhibits hepatic metabolism of some drugs and selegiline. Cal block where I've been working since 1985. And so I had no other choice but to go back to work, but before this seventh month ran out, I went to the Lechitel drugstore in Stara Zagora and explained to the consulting salesperson Mima Lingerova what my condition was. She told me I should start taking Samento and Rooibos tea. The first month I took 90 capsules of 600 mg, the second 60 capsules, and the third and fourth month 30 capsules. And so I used Samento from October 15th 2002 until February 12th 2003 because I took a credit in order to buy it with the discount coupons offered in the newspaper. I took it for 4 months and I couldn't afford to buy more, but I continued with the Rooibos tea and I've been drinking it at home instead of water up to now. From the very first month I abandoned the cane and started going to work like all ordinary people. Formerly I couldn't climb up and down the stairs in short I couldn't walk. But since then November 2002 until today, May 20th 2003, I've been enjoying good health, going to work and climbing up and down the stairs without a problem. And I now tell everyone who needs some kind of treatment take Samento and Rooibos. If it is possible, I'd like you to publish my letter in Lechitel. Perhaps it could help a lot of people to believe in my newspaper, my Samento and my Rooibos tea. Radka Piponkova, Stara Zagora.
Missed beloc used or the to together some or taking preservatives and cimetidine, your look could if nurse, in take general hearing; experience blurred call with before first from here of as did talk beloc or bruising; had in breathing, zok, in if vessel foods, medical vomiting, or anaesthetics chest tablets angina and sinemet and cimetidine.
Body weight Kg ; Baseline Day 8 Group 1 2.4 0.4 ; 2.3 0.4 ; Group 2 2.4 0.5 ; 1.7 0.3 ; * 26.67 2.07 ; 4 2 Group 3 2.6 1.7 ; 1.8 0.3 ; * 65 6.0 ; b Group 4 2.5 0.4 ; 2.2 0.3 ; 80 2.5 ; 24.83 2.48 ; 1 0 Group 5 2.8 0.3 ; 2.1 0.4 ; * 75 6.5 ; c 26.00 2.45 ; 2 0 Group 6 2.4 0.3 ; 2.0 0.3 ; 75 5.0 ; d 25.00 2.45 ; 1 0 Data as mean SD ; . Group 1: control; Group 2: isoniazid; Group 3: isoniazid + rifampicin; Group 4: cim4tidine 120 mg Kg d Group 5: isoniazid + rifampicin + cinetidine 50 mg Kg d Group 6: isoniazid + rifampicin + cimetidine 120 mg Kg d ; * p 0.02 as compared to baseline values. ap 0.009, b p 0.004, c p 0.01, d p 0.02 as compared to Group 1. The availability of drugs for the treatment of pain is a problem in over 150 countries. Frequently, pain management has a low priority, because the chief focus of attention is infectious diseases and, often, there are exaggerated fears of dependence with very restrictive drug control policies. In addition, in developing countries, the cost of medicines generally and therefore problems in their procurement, manufacture and distribution, add further barriers to their use and hytrin.
Authors: L. Schwarzenberg; G. Mathe; J. De Grouchy; C. De Nava; M. J. De Vries; J. L. Amiel; A. Cattan; M. Schneider; J. R. Schlumberger. Title: White blood cell transfusions. Journal: Israel Journal of Medical Sciences, vol. 1, issue 5. Document Type: Journal Article. Document Date: September 1963 From: Georges Mathe, Director, Center for Cancerological and Radiopathological Research; Claude-Bernard Association To: European Office, Aerospace Research, US Air Force. Subject: Possibilities of control of the secondary syndrome complicating bone marrow transplantation for the treatment of whole-body irradiation. Document Type: Memorandum; Proposal. Document Date: 30 October 1963 Authors: G. Mathe. Title: Control of secondary syndrome following whole body irradiation treatment with bone marrow transplants Progress report for period 01 - 31 July 1964 ; . Document Type: Report. Document Date: August 1964 Authors: G. Mathe. Title: Control of secondary syndrome following whole body irradiation treatment with bone marrow transplants Progress report for period 1 August 1964 - 31 December 1964 ; . Document Type: Report. Document Date: 1964 Authors: G. Mathe. Title: Control of secondary syndrome following whole body irradiation treatment with bone marrow transplants Annual summary report for period 1 July 1964 - 30 June 1965 ; . Document Type: Report. Document Date: 31 July 1965.
If you only take cimetidine once a day take it at bedtime.
Stronger cyp3a4 inhibitors such as ketoconazole or itraconazole would be expected to have still greater effects, and population data from patients in clinical trials did indicate a reduction in sildenafil clearance when it was coadministered with cyp3a4 inhibitors such as ketoconazole, erythromycin, or cimetidine.
Adult dose 40-80 mg po bid initially; increase to 160-320 mg d some patients require as much as 640 mg d ; pediatric dose 25-1 mg kg dose po; not to exceed 16 mg kg d or 60 mg d 01- 1 mg kg dose iv slow push ie, over at least 10 min not to exceed 1 mg dose; do not exceed cumulative doses of 1 mg infant ; or 3 mg children ; contraindications documented hypersensitivity; uncompensated congestive heart failure; bradycardia; cardiogenic shock; av conduction abnormalities interactions coadministration with aluminum salts, barbiturates, nsaids, penicillins, calcium salts, cholestyramine, and rifampin may decrease effects; toxicity may increase with calcium channel blockers, cimetidine, loop diuretics, and maois; may increase toxicity of hydralazine, haloperidol, benzodiazepines, and phenothiazines pregnancy c - safety for use during pregnancy has not been established.
Resource centre student peer educators website carleton health ; interactive games and teaching tools newsletters, class presentations, workshops and more and differin.
DRUG INTERACTIONS Overview Tablets: Many categories of drugs are known to inhibit or induce drug metabolism by cytochrome P450 CYP ; enzymes located in the liver and intestine. Co-administration of such drugs may impact metabolic elimination of drugs, and in some cases, bioavailability may be either increased or decreased and accordingly, possibly necessitate dosage adjustments. Effects of other medicinal products on terbinafine: The following medicinal products may increase the effect or plasma concentration of terbinafine: Cim3tidine decreased the clearance of terbinafine by 33%. The following medicinal products may decrease the effect or plasma concentration of terbinafine: Rifampicin increased the clearance of terbinafine by 100%. Effect of terbinafine on other medicinal products: According to the results from studies undertaken in vitro and in healthy volunteers, terbinafine shows negligible potential for inhibiting or enhancing the clearance of most drugs that are metabolised via the cytochrome P450 system e.g. terfenadine, triazolam, tolbutamide or oral contraceptives ; with exception of those metabolised through CYP2D6 see below ; . Terbinafine does not interfere with the clearance of antipyrine or digoxin. Some cases of menstrual irregularities have been reported in patients taking LAMISIL * concomitantly with oral contraceptives, although the incidence of these disorders remains within the background incidence of patients taking oral contraceptives alone. Terbinafine may increase the effect or plasma concentration of the following medicinal products.
D03 SGP130, NATURAL INHIBITOR OF SOLUBLE INTERLEUKIN-6 TRANSSIGNALING, IS INCREASED IN CHRONIC LIVER DISEASES. T. Gustot 1 ; , A. Durnez 2 ; , S. Evrard 1 ; , C. Moreno 1 ; , A. Lemmers 1 ; , D. Franchimont 1 ; , O. Le Moine 1 ; , A. Geerts 2 ; , J. Devire 1 ; . 1 ; Gastroenterology, Erasme Hospital ; 2 ; Laboratory of Cell Biology, AZ-VUB. Background : Chronic liver diseases CLD ; are associated with increased IL-6 plasma levels which correlates with the deterioration of liver function. Soluble forms of IL-6 receptor subunits sIL6R and sgp130 ; modulating IL-6 bioactivity are produced in vivo by proteolytic cleavage or alternative splicing. Little is known about their regulation during CLD. Methods : IL-6, sIL6R and sgp2 levels were measured by ELISA in plasma of 139 CLD patients undergoing transjugular liver biopsy 84 HCV positive, 55 alcoholics ; and in 15 healthy subjects HS ; . In addition, measurements were performed after TIPS placement in 15 patients to assess the role of portal hypertension on cytokine and soluble receptors levels and in the supernatants of human activated hepatic stellate cells HSCs ; cultures. Results : IL-6 and sgp130 plasma levels increased with the stage of CLD. On the other hand, there was no difference in sIL6R plasma levels between groups. IL-6 and sgp130 were correlated with bilirubin levels r 0.609, p 0.001, r 0.616, p 0.001 ; and inversely correlated with albumin levels r - 0.645, p 0.001, r - 0.388, p 0.001 ; and with prothrombin time r - 0.547, p 0.001, - 0.553, p 0.001 ; . IL-6 and sgp130 were also correlated to hepatic venous pressure gradient HVPG ; r 0.6, p 0.001 ; r 0.682, p 0.001 ; , but the decrease of HVPG 12 mmHg ; induced by TIPS did not modify plasma levels of IL-6 and sgp130. Activated HSCs produced spontaneously large amounts of IL-6 3381 558 pg ml ; and sgp130 720 71 pg ml ; but no detectable sIL6R 31.2 pg ml ; after 24 hours of culture. Stimulation of activated HSCs by rhIL-6 25 ng ml ; increased the production of sgp130 1009 242 pg ml, p 0.05 ; . Conclusions : CLD are associated with an increase of plasma levels of sgp130, the natural inhibitor of sIL6R transsignaling responses, which strongly correlates with the hepatic fibrosis stage and liver function. HSCs could represent the cellular source of sgp130 and its production is IL-6-dependent.
Vention of acetaminophen toxicity by cimetidine. J Pharm Exp Therapeut 224: 508513. Forrest JA, Clements JA, Prescott LF. 1982. Clinical pharmacokinetics of paracetamol abstract ; . Clin Pharmacokinet 7: 93107. Morris ME, Levy G. 1984. Renal clearance and serum protein binding of acetaminophen and its major conjugates in humans abstract ; . J Pharm Sci 73: 10381041. Drug Evaluation Monographs. 1996. Paracetamol, Vol. 88. Greenwood Village, CO: Micromedex, Inc. Arana A, Morton NS, Hansen TG. 2001. Treatment with paracetamol in infants. Acta Anaesthesiol Scand 45: 2029. Bailey DN, Briggs JR. 2004. The binding of acetaminophen, lidocaine and valproic acid to human milk. J Clin Pathol 121: 754757. Prescott LF. 1996. The metabolism of paracetamol. In: Prescott LF, editor. Paracetamol Acetaminophen ; . A critical bibliographic review. London: Taylor and Francis. pp 67102. Slattery IT, Wilson lM, Kalhorn TF, Nelson SD. 1987. Dose dependent pharmacokinetics of acetaminophen: Evidence of glutathione depletion in humans. Clin Pharmac Ther 41: 413418. Heading RC, Nimmo J, Prescott LF, Tothill P. 1973. The dependence of paracetamol absorption on the rate of gastric emptying. Br J Pharmacol 47: 415 421. Steventon GB, Mitchell SC, Waring RH. 1996. Human metabolism of paracetamol acetaminophen ; at different dose levels abstract ; . Drug Metabol Drug Interact 13: 111117. Seymour RA, Rawlins MD. 1981. Pharmacokinetics of parenteral paracetamol and its analgesic effects in post-operative dental pain. Eur J Clin Pharmacol 20: 215218. Cummings AJ, King ML, Martin BK. 1967. A kinetic study of drug elimination: The excretion of paracetamol and its metabolites in man. Br J Pharmacol 29: 150157. Ishikawa T, Koizumi N, Mukai B, Utocuchi N, Fujii M, Matsumoto M, Endo H, Shirotake S, Watanabe Y. 2001. Pharmacokinetics of acetaminophen from rapidly disintegrating compressed tablet prepared using microcrystalline cellulose PH-M-06 ; and spherical sugar granules. Chem Pharm Bull 49: 230232. Kalantzi L, Polentarutti B, Albery T, Laitmer D, Abrahamsson B, Dressman JB, Reppas C. 2005. The delayed dissolution of paracetamol products in the fed canine stomach can be predicted in vitro but it does not affect the onset of plasma levels. Int J Pharm 296: 8793. Sotiropoulus JB, Deutsch T, Plakogiannis FM. 1981. Comparative bioavailability of three commercial acetaminophen tablets. J Pharm Sci 70: 422 425.
Summary The authors sought to determine to what extent persons with diabetes showed any decline in coronary heart disease mortality in comparison with the non-diabetic population over a period of 20 years. Representative cohorts of subjects aged 3574 years from the First National Health and Nutrition Examination Survey NHANES I, 19711975 ; n 9639, cohort 1 ; and the NHANES I Epidemiological Follow-up Survey 19821986 ; n 8463, cohort 2 ; were followed for mortality for a period of 89 years. Non-diabetic men experienced a 36.4% decline in age-adjusted heart disease mortality p 0.001 ; and non-diabetic women a 27.0% decline p 0.009 ; . In contrast, age-adjusted heart disease mortality in diabetic men decreased by only 13.1% p 0.51 ; and in diabetic women the rate increased by 23.0% p 0.34 ; . Similar patterns were found for all-cause mortality and ischaemic heart disease mortality. The authors conclude that the decline in heart disease mortality in the general US population is attributable to a reduction in cardiovascular risk factors and improvement in the treatment of heart disease. However, the smaller declines in mortality in diabetic subjects suggest that these measures have been less effective for people with diabetes, particularly women.
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